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dc.contributor.authorMaurel, Marion
dc.contributor.authorSamali, Afshin
dc.contributor.authorChevet, Eric
dc.date.accessioned2018-09-20T16:16:19Z
dc.date.available2018-09-20T16:16:19Z
dc.date.issued2014-09-01
dc.identifier.citationMaurel, Marion; Samali, Afshin; Chevet, Eric (2014). Endoplasmic reticulum stress: at the crossroads of inflammation and metabolism in hepatocellular carcinoma development. Cancer Cell 26 (3), 301-303
dc.identifier.issn1535-6108
dc.identifier.urihttp://hdl.handle.net/10379/12687
dc.description.abstractSteatohepatitis is a cause of hepatocellular carcinoma development; however, the underlying mechanisms are poorly defined. In this issue of Cancer Cell, Nakagawa and colleagues demonstrate that activation of endoplasmic reticulum stress signaling is instrumental in the development of steatohepatitis and synergizes with proinflammatory pathways to promote hepatocarcinogenesis.
dc.publisherElsevier BV
dc.relation.ispartofCancer Cell
dc.rightsAttribution-NonCommercial-NoDerivs 3.0 Ireland
dc.rights.urihttps://creativecommons.org/licenses/by-nc-nd/3.0/ie/
dc.subjectdisease
dc.subjectactivation
dc.subjectpathway
dc.subjectgenes
dc.titleEndoplasmic reticulum stress: at the crossroads of inflammation and metabolism in hepatocellular carcinoma development
dc.typeArticle
dc.identifier.doi10.1016/j.ccr.2014.08.007
dc.local.publishedsourcehttps://doi.org/10.1016/j.ccr.2014.08.007
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Attribution-NonCommercial-NoDerivs 3.0 Ireland
Except where otherwise noted, this item's license is described as Attribution-NonCommercial-NoDerivs 3.0 Ireland